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Clinics & Services Resources
There is evidence that an increase in plasma
homocysteine may increase the formation of asymmetric dimethylarginine
(ADMA), an inhibitor of nitric oxide synthase. The methyl
groups within ADMA are derived from the conversion of S-adenosylmethionine
to S-adenosylhomocysteine intermediates in the methionine/homocysteine
pathway.
No previous study has assessed the role of
methylation status, its impact on ADMA formation, and their
association with endothelial function in healthy human beings.
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